Meirong Bai

Meirong Bai

About Meirong Bai

Meirong Bai is a scientist specializing in the physiological response to hypoxia, hypothermia, and mitochondrial stress. Currently working at Alamar Biosciences, Inc., Bai aims to develop novel therapies for ischemic and metabolic diseases and age-related neurodegeneration.

Work at Alamar Biosciences

Meirong Bai has been employed as a Scientist at Alamar Biosciences, Inc. since 2022. In this role, Bai focuses on research aimed at discovering novel therapeutic approaches for treating ischemic and metabolic diseases, as well as age-related neurodegeneration. The work at Alamar Biosciences involves applying advanced scientific techniques to address significant health challenges.

Education and Expertise

Meirong Bai holds a Bachelor's degree in Biology/Biological Sciences from East China Normal University, where studies spanned from 2006 to 2011. Bai also pursued a secondary major in Multimedia and Network Technology at the same institution. Furthering academic qualifications, Bai earned a Doctor of Philosophy (Ph.D.) in Molecular Biology from Fudan University. This educational background provides a strong foundation for Bai's current research endeavors.

Background

Meirong Bai's research career includes significant experience at the University of California, San Francisco (UCSF). Bai worked as a Postdoctoral Fellow from 2019 to 2021, contributing to research in the San Francisco Bay Area. During this time, Bai conducted studies on the physiological responses to hypoxia, hypothermia, and mitochondrial stress, under the mentorship of Dr. Dengke Ma at the UCSF Cardiovascular Institute.

Research Focus

Bai's research primarily investigates the physiological responses to conditions such as hypoxia and hypothermia, as well as the effects of mitochondrial stress. This work is critical in understanding how these factors contribute to various health conditions. The aim is to develop therapeutic strategies that can effectively address ischemic and metabolic diseases, along with age-related neurodegeneration.

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