June Yong Lee
About June Yong Lee
June Yong Lee is a Postdoctoral Research Fellow at NYU Langone Health with a focus on the pathogenic Type-17 immune program and its implications in chronic inflammatory diseases and epithelial carcinoma.
Current Position at NYU Langone Health
June Yong Lee is a Postdoctoral Research Fellow at NYU Langone Health. In this role, he focuses on advanced research in the field of immunology, primarily investigating chronic inflammatory diseases. His current studies involve the Type-17 immune program, exploring how it contributes to disease pathology and understanding its implications in epithelial carcinoma.
Previous Experience at University of Minnesota
June Yong Lee served as a PhD Graduate Student at the University of Minnesota from 2010 to 2014. During his time there, he conducted significant research on the role of Serum Amyloid A protein family in chronic inflammatory diseases, particularly through mouse models of colitis and neuroinflammation. His doctoral work culminated in earning a PhD in Immunology.
Educational Background
June Yong Lee holds a Doctor of Philosophy (PhD) in Immunology from the University of Minnesota-Twin Cities, completed in 2014. He also earned a Master of Science (MS) in Immunology from Korea University in 2008 and a Bachelor of Science (BS) in General Biology from the same institution in 2006. His extensive academic training has provided a strong foundation for his research in chronic inflammatory diseases.
Published Research and Contributions
June Yong Lee has made notable contributions to the field of immunology through his research publications. In 2019, he published a study in 'Cell' on the role of Serum Amyloid A proteins in chronic inflammatory diseases. Additionally, he co-authored a significant paper in 'Genome Research' that identified ROR-alpha as a key regulator in maintaining the Th17 lineage in inflamed tissues. He is currently preparing a manuscript on the transcriptional regulatory network of Th17 cells.
Research Focus
June Yong Lee's research primarily focuses on the pathogenic Type-17 immune program and its implications in chronic inflammatory diseases and epithelial carcinoma. His work aims to uncover the underlying mechanisms of immune responses in these conditions, with a particular interest in the role of the Serum Amyloid A protein family and Th17 cells. His research has the potential to inform new therapeutic approaches for treating chronic inflammatory diseases.